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Nicotinamide Mononucleotide Alleviates Aging Defects in Hutchinson–Gilford Progeria Syndrome - Xu - 2025 - The FASEB Journal - Wiley Online Library
Kang MS, Park GY, Lee AR
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Furthermore, interventional studies in DSS-colitis suggest targeting ferroptosis could be therapeutic: β-caryophyllene inhibition of macrophage lipid peroxidation and ferroptosis was linked to suppression of inflammation [12], and seliciclib-treated mice—that displayed reduced epithelial ferroptosis—had less severe DSS-induced colitis [41]
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