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486 Overexpression of GPX4 in cardiomyocytes or targeting the mitochondrial DOX-Fe 2+ complex prevents DOX-induced ferroptosis and improves cardiomyocyte injury
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In IPF patients, neutrophils show an increased expression of Nox2 subunits (Kato and Hecker, 2020) and genetic removal of some of these subunits partially protects from the development of fibrosis in lung fibrosis mice models (Manoury et al., 2005
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